Inhibitory postsynaptic peev xwm (IPSPs) yog txuam nrog transmitter-activated influx ntawm Cl− thiab membrane hyperpolarization.
Dab tsi neurotransmitter ua rau inhibitory postsynaptic muaj peev xwm?
Tso tawm cov neurotransmitter ntawm inhibitory synapses ua rau inhibitory postsynaptic peev xwm (IPSPs), ib qho hyperpolarization ntawm presynaptic membrane. Piv txwv li, thaum lub neurotransmitter GABA (gamma-aminobutyric acid) raug tso tawm los ntawm presynaptic neuron, nws khi rau thiab qhib Cl– channels.
Dab tsi ions cuam tshuam nrog IPSP inhibitory postsynaptic muaj peev xwm)?
Conversely, inhibitory postsynaptic muaj peev xwm (IPSPs) tshwm sim los ntawm kev nkag mus ntawm tsis zoo ions (piv txwv li, Cl−) rau hauv, lossis cov efflux ntawm cov ions zoo (piv txwv li, K +), tawm ntawm postsynaptic cell.
Postynaptic inhibition tshwm sim los ntawm dab tsi?
Raws li kev pom zoo, postsynaptic inhibition induced los ntawm qhov ua kom ntawm GABA Athiab glycine receptors muaj nyob rau hauv ob lub tswv yim: shunting incoming excitatory tam sim no thiab txav cov membrane muaj peev xwm deb ntawm qhov kev txiav txim muaj peev xwm pib.
Dab tsi ua rau inhibitory muaj peev xwm?
A depolarizing inhibitory postsynaptic muaj peev xwm yog vim synaptic tam sim no uas nws qhov kev thim rov qab yog qhov zoo dua li qhov muaj peev xwm so ntawm daim nyias nyias tab sis ntau qhov tsis zoo tshaj qhov pib rau kev qhib lub Na+ channels of the action potential.
