Caffeine nce lub zog metabolism thoob plaws hauv lub hlwb tab sis txo qis tib lub sijhawm cerebral ntshav ntws, ua rau cov txheeb ze lub hlwb hypoperfusion. Caffeine activates noradrenaline neurons thiab zoo li cuam tshuam rau lub zos tso tawm ntawm dopamine.
Caffeine ua dab tsi rau neurotransmitters?
Adenosine txo qis tus nqi hluav taws xob neuronal thiab inhibits kev sib kis ntawm synaptic thiab tso tawm ntawm cov neurotransmitters feem ntau. Caffeine kuj nce kev hloov pauv ntawm ntau cov neurotransmitters, suav nrog monoamines thiab acetylcholine.
Caffeine cuam tshuam txog homeostasis li cas?
Feem ntau tsom rau adenosine receptors, caffeine ua rau hloov pauv hauv qabzib homeostasis los ntawm kev txo qis cov piam thaj mus rau hauv cov leeg pob txha, yog li ua rau nce hauv cov ntshav qabzib concentration.
Yuav ua li cas yog lub tshuab ua haujlwm rau caffeine?
Caffeine kev txiav txim yog xav tias yuav kho los ntawm ntau lub tswv yim: kev tawm tsam ntawm adenosine receptors, inhibition ntawm phosphodiesterase, tso tawm calcium los ntawm cov khw muag khoom intracellular, thiab antagonism ntawm benzodiazepine receptors (Myers li al., 1999).
Caffeine khi rau adenosine li cas?
Adenosine yog lub hauv paus paj hlwb neuromodulator uas muaj cov receptors tshwj xeeb. Thaum adenosine khi rau nws cov receptors, kev ua haujlwm ntawm neural qeeb qis, thiab koj tsaug zog. … Caffeine ua raws li adenosine-receptor antagonist. Qhov no txhais tau hais tias nws khi rau cov tib receptors, tab sis tsis txo cov kev ua ntawm neural.
