Insulin hloov cov poov tshuaj rau hauv cov hlwb los ntawm kev txhawb nqa kev ua haujlwm ntawm Na + -H + antiporter ntawm cell membrane, txhawb kev nkag mus ntawm sodium rau hauv cov hlwb, uas ua rau ua kom cov Na + -K + ATPase, ua rau cov electrogenic influx ntawm poov tshuaj. IV insulin ua rau ib koob tshuaj - dependent poob ntawm cov ntshav hauv cov poov tshuaj [16].
Vim li cas insulin ua rau cov poov tshuaj tsawg?
Exogenous insulin tuaj yeem induce me hypokalemia vim nws txhawb kev nkag mus ntawm K+ rau hauv cov leeg pob txha thiab cov hlwb los ntawm kev nce cov haujlwm ntawm the Na+-K+-ATPase pump[39]. Kev tso tawm ntau ntxiv ntawm epinephrine vim yog insulin-induced hypoglycemia kuj tseem tuaj yeem ua lub luag haujlwm pabcuam [40].
Puas insulin txo cov poov tshuaj?
Insulin pab txav cov poov tshuaj mus rau hauv hlwb. Qhov no tuaj yeem ua rau hypokalemia, lossis qis poov tshuaj hauv cov ntshav.
insulin txo cov poov tshuaj ntau npaum li cas?
Insulin 10 units yog kwv yees kom txo cov poov tshuaj hauv cov ntshav los ntawm 0.6–1.2 mMol/L hauv 15 feeb kev tswj hwm nrog cov teebmeem ntev 4-6 teev (1 –3). Txawm li cas los xij, insulin kuj tseem tuaj yeem ua rau muaj qhov tsis xav tau, xws li hypoglycemia (1, 2.
insulin thiab piam thaj kho li cas hyperkalemia?
Tshuaj siv rau kev kho mob hyperkalemia suav nrog cov hauv qab no: Calcium (xws li gluconate lossis chloride): Txo qhov kev pheej hmoo ntawm ventricular fibrillation los ntawm hyperkalemia. Cov tshuaj insulin siv nrog cov piam thaj: Ua kom cov piam thaj nkag mus rau hauv lub cell, uas ua rau muaj kev hloov pauv ntawm cov poov tshuaj hauv lub cev.
