Mechanism of Action Aminocaproic acid yog lysine analog uas khi kev sib tw rau plasminogen, thaiv plasminogen los ntawm kev khi rau fibrin thiab tom qab hloov mus rau plasmin Qhov kev ua no tom qab ua rau inhibition ntawm fibrin degradation (fibrinolysis).[4][5]
aminocaproic siv rau dab tsi?
Aminocaproic acid yog siv los kho mob ntuavrau cov neeg muaj qee yam mob xws li aplastic anemia (tsis muaj cov qe ntshav thiab platelets), cirrhosis ntawm daim siab, placenta abruptio (Kev sib cais thaum ntxov ntawm cev xeeb tub), tso zis los ntshav, thiab qee yam mob qog noj ntshav.
Yuav ua li cas yog lub mechanism ntawm kev txiav txim ntawm aminocaproic acid?
Mechanism of Action
Aminocaproic acid inhibits ob txoj haujlwm ntawm plasminogen activators thiab mus rau qib qis dua, plasmin kev ua los ntawm kev khi rau lysine-binding qhov chaw hauv plasminogen / plasmin molecule, uas cuam tshuam nrog lub peev xwm ntawm plasmin rau lyse fibrin clots.
Amicar lub hom phiaj yog dab tsi?
Cov tshuaj no tau siv. Qhov no tuaj yeem ua rau los ntshav hnyav tom qab qee qhov kev phais lossis qee yam mob (xws li los ntshav, kab mob siab, mob qog noj ntshav).
Nws siv sijhawm ntev npaum li cas rau aminocaproic acid ua haujlwm?
Cov teebmeem fibrinolysis-inhibitory ntawm AMICAR zoo li tau tawm dag zog los ntawm inhibition ntawm plasminogen activators thiab qis dua los ntawm kev ua haujlwm antiplasmin. Hauv cov neeg laus, qhov nqus nqus ntawm qhov ncauj zoo li yog qhov kev txiav txim xoom nrog tus nqi nqus ntawm 5.2 g / h. Lub sij hawm nruab nrab lub sij hawm hauv nqus yog 10 feeb
