Kev txiav txim siab txuas ntxiv lossis txiav kev siv ACEi/ARB thaum cov neeg mob mus txog CKD theem 4 lossis 5 yog qhov tsis txaus ntseeg. Ntawm ib sab, kev pheej hmoo cuam tshuam nrog kev txuas ntxiv suav nrog hyperkalemia, metabolic acidosis, thiab txo qis hauv GFR.
Ntawm li cas creatinine qib yuav tsum ACE inhibitors raug tso tseg?
Cov kws sau ntawv pom zoo tias kev kho ACE inhibitor yuav tsum tsis txhob txiav tawm tshwj tsis yog serum creatinine qib nce siab dua 30% dhau qhov pib hauv thawj 2 lub hlis tom qab pib ntawm kev kho lossis hyperkalemia (cov ntshav poov tshuaj. qib > lossis=5.6 mmol / L) txhim kho.
Vim li cas ACE inhibitors yog contraindicated hauv CKD?
Qhov kev txhawj xeeb loj txog kev nyab xeeb nrog ACE-inhibitor lossis ARB kev kho mob hauv tus neeg mob CKD yog hyperkalemia thiab poob sai hauv GFR. Cov tshuaj no yuav tsum tsis txhob siv rau cov neeg mob uas muaj kab mob hyperkalemia.
Koj puas tuaj yeem siv ACE inhibitors hauv CKD?
ACE inhibitors thiab ARBs tuaj yeem siv tau nyab xeeb rau feem ntau cov neeg mob nrog CKD. 11.1 ACE inhibitors thiab ARBs yuav tsum tau siv ntawm qhov nruab nrab mus rau qhov siab, raws li siv hauv kev sim tshuaj) (A).
Thaum twg koj siv ACEi hauv CKD?
Angiotensin-hloov enzyme inhibitors (ACEIs) lossis angiotensin II receptor blockers (ARBs) yog cov tshuaj tiv thaiv ntshav siab zoo tshaj plaws uas muab kev tiv thaiv rau lub raum thiab cov hlab plawv tseem ceeb rau cov neeg mob CKD [9], thiab tau raug pom zoo los ua thawj kab kev kho mob rau cov neeg mob uas tsis yog mob ntshav qab zib CKD , tshwj xeeb…
