Yog li, cov nyhuv ntawm catecholamines ua rau cov ntshav qabzib nce nrawm(Thurston li al., 1993). Catecholamines (NE thiab suav tias yog E) kuj txhawb nqa gluconeogenesis ntawm α-adrenergic receptor ua rau ntawm intracellular calcium mobilization (Cramb li al., 1982).
Yuav ua li cas cov tshuaj hormones yuav ua rau cov ntshav qabzib ntau ntxiv?
Insulin thiab glucagon ua haujlwm hauv lub voj voog. Glucagon cuam tshuam nrog lub siab kom nce ntshav qab zib, thaum insulin txo cov ntshav qab zib los ntawm kev pab cov hlwb siv cov piam thaj.
YPob catecholamines nce glucagon?
Glucagon thiab catecholamines qhia ntau yam teebmeem hauv metabolic. Tsis tas li ntawd, catecholamines yog muaj zog stimulators ntawm glucagon secretionthiab, nyob rau hauv tej yam kev mob, glucagon stimulates catecholamine tso tawm ntawm adrenal medulla.
YPuas catecholamines nce insulin?
Catecholamines stimulate insulin tso tawm los ntawm stimulation ntawmpancreatic β receptor.
Catecholamines txhawb dab tsi?
Catecholamines ua rau muaj kev hloov pauv ntawm lub cev uas npaj lub cev rau kev ua si lub cev (qhov kev sib ntaus sib tua lossis kev sib ntaus sib tua). Qee qhov teebmeem tshwm sim yog nce hauv lub plawv dhia, ntshav siab, ntshav qabzib ntau, thiab cov tshuaj tiv thaiv ntawm lub paj hlwb sympathetic.
