Kev tshuaj ntsuam ntawm 240 tus tub ntxhais kawm lag ntseg-mute tau qhia tias qhov laj thawj tseem ceeb ntawm kev lag ntseg hauv lub hauv paus muaj tau los ntawm(68.5%) uas txawv ntawm qhov ua ntej 1970s. Ntawm cov neeg mob uas muaj kev lag ntseg qeeb, 29.8% yog keeb kwm.
Yuav ua li cas rau ib tug neeg lag ntseg thiab hais lus?
Neeg-mute yog ib lo lus uas tau siv keeb kwm los txheeb xyuas tus neeg lag ntseg thiab siv hom lus lossis lag ntseg thiab hais tsis tau.
Tus neeg lag ntseg-tsis mloog tau?
Kev phem. tsis tau hnov thiab hais. ib tug neeg uas hnov thiab hais tsis tau lus, tshwj xeeb tshaj yog ib tug neeg uas hais lus tsis tau yog vim lub hauv paus los yog lag ntseg thaum ntxov.
Yuav ua li cas lag ntseg yog caj ces?
Kwv yees li 80% ntawm prelingual lag ntseg yog caj ces, feem ntau autosomal recessive thiab nonsyndromic. Feem ntau ua rau mob hnyav-rau-qhov tob autosomal recessive nonsyndromic hnov lus tsis zoo hauv cov neeg feem coob yog kev hloov pauv ntawm GJB2.
Puas lag ntseg hauv tsev neeg?
Puas hnov tsis hnov lus hauv tsev neeg? Yog, hnov tsis hnov lus tuaj yeem cuam tshuam cov tswv cuab ntawm tib tsev neeg. Cia peb tham txog qee yam ntawm qhov no tuaj yeem tshwm sim. Ib txoj hauv kev tsis hnov lus tuaj yeem cuam tshuam rau tsev neeg yog los ntawm caj ces.
