Ib qho ntawm cov tsos mob classical ntawm pheochromocytoma ntsoog yog hyperglycemia [1] uas yuav tshwm sim los ntawm nce insulin tsis kam nyob rau hauv cov ntaub so ntswg peripheral thiab impaired insulin secretion[2].
Vim li cas pheochromocytoma ua rau hyperglycemia?
Hyperglycemia tau txuas rau qhov hnyav dua ntawm insulin tsis kam tshwm sim los ntawm catecholamine ntau dhauhauv pheochromocytoma. Cov nyhuv inhibitory ntawm catecholamines ntawm insulin secretion yog xav tias yuav kho los ntawm α-2 adrenergic receptors.
Puas pheochromocytoma ua rau hypoglycemia?
Pheochromocytoma, qog nqaij hlav uas tshwm sim los ntawm catecholamine ntau dhau, feem ntau cuam tshuam nrog kev ua haujlwm tsis zoo ntawm cov piam thaj. Hypoglycemia tuaj yeem tshwm sim tom qab tshem tawm sai sai ntawm catecholamines hauv lub sijhawm tom qab phais.
Vim li cas pheochromocytoma ua rau cem quav?
Yog li ntawd, siab ntau ntawm catecholaminesyuav ua rau txo qis hauv plab hnyuv peristalsis, motility, thiab lub suab. Clinically qhov no yuav tshwm sim thawj zaug raws li cem quav, tab sis thaum cov qib catecholamine nce siab ntxiv, lawv tuaj yeem ua rau cov ileus los yog tej zaum megacolon.
YHyperglycemia tshwm sim li cas?
hyperglycemia yog dab tsi? Hyperglycemia, lossis ntshav qabzib siab, tshwm sim thaum muaj piam thaj ntau dhau hauv cov ntshavQhov no tshwm sim thaum koj lub cev muaj insulin tsawg dhau (cov tshuaj hormones thauj cov piam thaj mus rau hauv cov ntshav), lossis yog tias koj lub cev muaj cov tshuaj insulin tsawg dhau. Lub cev tsis tuaj yeem siv insulin kom raug. Tus mob feem ntau cuam tshuam nrog ntshav qab zib.
