Phase II thaiv Nws tshwm sim tom qab rov ua dua boluses lossis lub sijhawm ntev ntawm succinylcholine. Hauv cov neeg mob atypical plasma cholinesterase, Phase II thaiv tuaj yeem txhim kho tom qab ib koob tshuaj.
Vim li cas Phase 2 Block tshwm sim?
Mechanism ntawm Phase II Block
Qhov no yog vim nce kev ua haujlwm ntawm sodium-potassium ATPase twj tso kua mis, uas coj cov poov tshuaj rau hauv lub cell pauv rau sodium. Cov receptor tsis teb kom tsim nyog rau acetylcholine, thiab neuromuscular blockade yog ncua ntev.
hom tshuaj twg yog rocuronium?
Rocuronium yog tsis yog-depolarizing neuromuscular blocker uas yog siv dav los tsim cov leeg nqaij kom pab txhawb kev phais thiab ua pa ntawm lub ntsws hauv cov xwm txheej xaiv thiab tshwm sim.
Cov tshuaj tiv thaiv neuromuscular yog dab tsi?
Neuromuscular blocking agents (NMBAs) tuaj nyob rau hauv ob daim ntawv: depolarizing neuromuscular blocking agents (xws li, succinylcholine) thiab nondepolarizing neuromuscular blocking agents (piv txwv li, rocuronium, vecuronium, atracurium, ci., mivacurium).
YPuas neostigmine thim rov qab succinylcholine?
Nws tau txiav txim siab tias succinylcholine-induced theem II thaiv tuaj yeem nyab xeeb thiab nrawm nrawmnrog neostigmine.
