Muaj ob hom kev thauj cov piam thaj hauv lub hlwb: cov piam thaj thauj cov protein (GLUTs) uas thauj cov piam thaj los ntawm kev yooj yim diffusion (a daim ntawv thauj mus los), thiab sodium- dependent glucose transporters (SGLTs) uas siv lub zog-coupled mechanism (active thauj).
Puas glut active thauj?
Muaj ob hom kev thauj cov piam thaj hauv lub hlwb: cov piam thaj thauj cov protein (GLUTs) uas thauj cov piam thaj los ntawm kev yooj yim diffusion (ib hom kev thauj mus los), thiab sodium-dependent glucose transporters (SGLTs) uas sivib lub zog-coupled mechanism (active thauj)
Yuav ua li cas thauj khoom yog tus thauj glut?
Tsev neeg GLUT lossis SLC2A yog tsev neeg cov protein uas pom muaj nyob hauv cov qe menyuam feem ntau. 14 GLUTS yog encoded los ntawm tib neeg genome. GLUT yog ib hom uniporter transporter protein.
Puas yog glut transporters carriers?
Glucose ua lub zog loj rau cov txheej txheem metabolic hauv cov hlwb mammalian. Txij li cov polar molecules tsis tuaj yeem thauj mus los hla cov plasma membrane, cov khoom thauj khoom hu ua glucose transporters yog xav tau rau cellular uptake.
Puas yog GLUT4 nquag thauj lossis tsis thauj khoom?
Insulin ua rau GLUT4 nkag mus rau hauv daim nyias nyias ntawm cov hlwb kom cov piam thaj tuaj yeem nkag mus hauv cov ntshav. Txij li qhov no yog passive mechanism, cov piam thaj nkag mus rau peb lub hlwb yog proportional rau ntau npaum li cas qab zib peb haus, mus txog rau lub point uas tag nrho peb cov channel raug siv (saturation).
