Hyperpolarization feem ntau tshwm sim los ntawm qhov txiaj ntsig zoo potassium K + (a cation) outflow los ntawm K + channels lossis Clˉ (ib qho anion) nkag los ntawm Clˉ raws. … Yog hais tias ib lub cell tau so Na + lossis Ca² + flux, inhibiting qhov tam sim no ntws kuj yuav ua rau hyperpolarization.
Dab tsi ua rau hyperpolarization?
Hyperpolarization feem ntau tshwm sim los ntawm efflux ntawm K+ (a cation) dhau K+ channels, los yog influx ntawm Cl – (ib qho anion) los ntawm Cl– channels. Ntawm qhov tod tes, influx ntawm cations, e.g. Na+ via Na+ channels or Ca2+ los ntawm Ca2+ channels, inhibits hyperpolarization.
Yuav ua li cas thaum lub neuron raug them zoo?
Thaum lub paj hlwb impulse (uas yog li cas cov neurons sib txuas lus nrog ib leeg) raug xa tawm los ntawm lub cev ntawm tes, cov sodium channels hauv cell membrane qhib thiab cov sodium cell nce mus rau hauv lub cellThaum lub xovtooj mus txog qhov chaw pib, qhov kev ua haujlwm muaj peev xwm yuav tua hluav taws, xa hluav taws xob teeb liab mus rau axon.
Yuav ua li cas cov channel ua rau hyperpolarization?
Hyperpolarization-activated thiab cyclic nucleotide-gated (HCN) channels belongs rau superfamily ntawm voltage-gated ion channels (1⇓–3). Ntawm hyperpolarization, HCN channels qhib thiab nqa ib qho Na+ sab hauv tam sim no uas tig mus depolarizes cell.
Dab tsi ua rau depolarization thiab hyperpolarization?
Depolarization thiab hyperpolarization tshwm sim thaum ion channels hauv daim nyias nyias qhib lossis kaw, hloov lub peev xwm ntawm cov ions tshwj xeeb nkag los yog tawm ntawm lub cell.… Qhov qhib ntawm cov channel uas cia cov ions zoo ntws tawm ntawm lub xov tooj (lossis cov ions tsis zoo ntws hauv) tuaj yeem ua rau hyperpolarization.
